Hemolytic anemia in mice przypominajš hereditary defects in humans. These defects umożliwiajš analyze the precise molecular basis of these diseases. Moreover, attempts to make pozwalajš gene therapy korygujšcej these defects, such as the procedure described by Doonera et al (Exp. Hematol. 2000, 28: 765-774). The techniques Dajs hope to use gene therapy to treat inherited human diseases, such as sickle cell anemia, thalassemia, sickle cell and eliptocytoza recessive.