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The infection caused by Helicobacter pylori is an important risk factor for gastric and duodenal ulcer. Infection with the bacterium causes a disturbance in the structure and function of cells of the gastric mucosa which may be the beginning of a number of pathological processes, including neoplastic transformation. Infection is associated with the development of chronic inflammation of the gastric epithelial cells of the microorganism caused by adaptation to the environment, defined by the chemical structure and specificity of the bacterial adhesins, the enzymatic activity of H. pylori urease, and its motility and chemotaxis. The work molecular basis of pathogenicity of bacteria concerns structural and functional diversity of the genome of H. pylori colonization and the mechanism of the gastric mucosa, the understanding of which is important both cognitive and clinical.
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The Editorial Board
Andrzej Łukaszyk - przewodniczący, Zofia Bielańska-Osuchowska, Szczepan Biliński, Mieczysław Chorąży, Aleksander Koj, Włodzimierz Korochoda, Leszek Kuźnicki, Aleksandra Stojałowska, Lech Wojtczak

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Katedra i Zakład Histologii i Embriologii Uniwersytetu Medycznego w Poznaniu, ul. Święcickiego 6, 60-781 Poznań, tel. +48 61 8546453, fax. +48 61 8546440, email:

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